By Eva Brehmer-Andersson
This ebook introduces the rules of microscopic pathology of received epidermis illnesses in a concise, but entire demeanour. The underlying mechanisms of the several illnesses are mentioned just about sleek wisdom approximately immunology, molecular biology, and inflammatory reaction to infections and damage. The booklet is amply illustrated and CD-ROM enables expansion of the illustrations.
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Extra resources for Dermatopathology
1979) introduced the concept of apoptosis in dermatopathology.
1a). A parallel phenomenon may appear with T cells as actors. In these cases a specific antigen present in the skin stimulates sensitized T cells to blast transformation and proliferation at the same place. , genetically identical T cells). These cells may be observed in herpes virus infection, vasculitis, and pityriasis lichenoides et varioliformis acuta (Figs. 3f). 5 Cytokines and Cell Adhesion Molecules Cytokines are cell-secreted soluble proteins which act as messenger molecules of the immune system.
These processes are progressive and continue for several hours after the cells have died. First, after about 12 h, signs of disintegration of the dead muscle cells become visible under the microscope as a necrotic area. If the patient survives, this area will be converted into a fibrotic scar after about 2 months. , the denaturation process prevails over the autolytic one), and gives the necrotic area a firm texture. This is in contrast to ischemic infarction in the brain, in which the tissue becomes soft due to a rich content of hydrolytic enzymes and in the end turns into a fluid-filled cavity (liquefaction necrosis).